Bridging Epigenomics and Pathology to Transform Discovery

We’ve partnered with 130+ labs worldwide

Explore real datasets, sample outputs, and visualizations from our spatial epigenomics workflows.

AtlasXomics Sample Qualification Assay White Paper

We have qualified FFPE clinical samples using our in-situ bulk assay, which reliably predicts full spatial assay success and helps investigators de-risk their studies.

Spatial profiling of chromatin accessibility in formalin-fixed paraffin-embedded tissues

Deng lab demonstrates spatial FFPE-ATAC with DBiT-seq, mapping chromatin accessibility in archived brain, thymus, and melanoma blocks at 10 µm resolution.

ASHG 2025 – Uncovering Tumor Resistance Mechanisms with Spatial CUT&Tag and Spatial ATAC-seq

Spatial CUT&Tag and ATAC-seq reveal pathology-aligned regulation and group-specific chromatin looping in gastric and prostate tumors.

Spatial profiling of chromatin accessibility reveals alteration of glial cells in Alzheimer’s disease mouse brain

AtlasXomics spatial ATAC-seq charts chromatin landscapes in AD-model mouse brains, uncovering microglia- and astrocyte-specific accessibility shifts tied to neuro-inflammation and synaptic dysfunction.

Spatial epigenomic niches underlie glioblastoma cell state plasticity

Spatial ATAC at 25 µm, combined with multi-omics and snATAC across 28 GBM resections, reveals a consistent layout with SOX10-high OPC-like “islands” nested within the tumor microenvironment.

Epigenomic landscape of the human dorsal root ganglion: sex differences and transcriptional regulation of nociceptive genes

Spatial ATAC-seq of human DRG uncovers X-linked EGR-motif enhancers enriched in female neurons and AP-1-driven Ca²⁺ pathways in males, outlining sex-specific pain signaling programs.

Basic-Leucine-Zipper Transcription Factors Regulate Selective Molecular Phenotypes in Regulatory T Cells During IL-2-Induced Activation

Spatial ATAC-seq at 20 µm pinpoints activated Treg niches in spleen and lung, with BATF/BACH1 motifs opening at sites matching Th-like phenotypes observed in bulk and single-cell data.